24 Nov 08 Tongat Ali, especially the LJ100, Increased Sex Drive
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You will be very pleased with my results of taking tongat ali, especially the LJ100. I have experienced tremendous improvements in tone and overall body mass. Above I linked the Wikipedia page that is actually very informative.
A big “side effect” that I have personally noticed is an increased sex drive. LJ100 ™ is an extremely high quality 100:1 standardized Tongkat Ali extract. We guarantee this product to be the highest quality LJ100.
Tongkat Ali is the popular folk name for Eurycoma Longifolia, a medium sized, slender rain forest tree. The name Tongkat Ali means Ali’s walking stick and the plant is native to Malaysia, lower burma, Thailand and Indonesia. Tongkat Ali enjoys a history of use that dates back to the 1700’s, and today there is a growing body of serious science that corroborates its traditional uses, specifically for the patented and proprietary brand LJ100 Tongkat Ali standardized extract containing 28% bioactive glycopeptides.
LJ100 Tongkat Ali in Testyx TPL
LJ100 is a proprietary, patented ingredient, and has become recognized as the premier brand of Eurycoma Longifolia for supplements that build and tone muscles, boost energy levels, decrease body fat, slow the aging process, and increase libido for health-conscious consumers. LJ100 has undergone an exclusive, patented extraction process to capture the most potent, biologically active compounds. SourceOne Global Partners, headquarters in Chicago, holds the exclusive distribution rights to market and sell LJ100 Tongkat Ali in dietary supplements.
ATP and Lean Muscle
In studies, LJ100 Tongkat Ali extract greatly increases ATP production. ATP, or adenosine triphosphate, is the basic unit of energy in the body, responsible for keeping us alive and going. By increasing ATP, overall energy and vitality are increased. Most people seek more energy and LJ100 Tongkat Ali provides it, without hyper stimulation, jittery nerves or insomnia. Promoting human energy production is a valuable health benefit by itself to make LJ100 Tongkat Ali an enduring botanical superstar. People want energy more than just about any other functional attribute.
Endocrinologists have known for a long time that testosterone increases the body’s ratio of lean muscle mass to fat. In both animals and humans, LJ100 Tongkat Ali increases muscle mass. In a study of men, half the subjects ingested LJ100 and half did not. In an eight-week physical training program the men who consumed LJ100 experienced greater gains in muscle mass and strength than those that did not. This demonstrates the powerful anabolic properties of Tongkat Ali. Instead of turning to the use of dangerous and potentially lethal steroids, it is recommended that more athletes opt for Tongkat Ali. In Malaysia, many professional field hockey players use LJ100 Tongkat Ali as an androgen and swear to its performance-enhancing effects. According to Chris Kilham, ethno botanist, author and lecturer, in a recent article in Physical Magazine,. “LJ100 Tongkat Ali has potential to revolutionize the sport nutrition category.”
Tags: LJ100, Testosterone, Testyx TPL, Tongkat Ali
24 Nov 08 Testosterone Stroke Research
Age-dependent effects of testosterone in experimental stroke
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This research was funded by US Public Health Service grants NS049210 and NS33668 and American Heart Association Grant 0825526G.
Jian Cheng1, Weidong Hu2, Thomas J Toung2, Zhizheng Zhang1, Susan M Parker1, Charles E Roselli1,3 and Patricia D Hurn1,3,4
- 1Department of Anesthesiology and Peri-Operative Medicine, Oregon Health and Science University, Portland, Oregon, USA
- 2Department of Anesthesiology and Critical Care Medicine, John Hopkins School of Medicine, Baltimore, Maryland, USA
- 3Department of Physiology and Pharmacology, Oregon Health and Science University, Portland, Oregon, USA
- 4Department of Neurology, Oregon Health and Science University, Portland, Oregon, USA
Correspondence: Professor PD Hurn, Department of Anesthesiology and Peri-Operative Medicine, Oregon Health and Science University, 3181 S.W. Sam Jackson Park Road, UHS-2, Portland, OR 97239-3098, USA. E-mail: hurnp@ohsu.edu
Received 12 August 2008; Revised 18 October 2008; Accepted 20 October 2008; Published online 12 November 2008.
Abstract
Although male sex is a well-recognized risk factor for stroke, the role of androgens in cerebral ischemia remains unclear. Therefore, we evaluated effects of testosterone on infarct size in both young adult and middle-aged rats (Wistar, 3-month versus 14-month old) and mice (C57/BL6, 3-month versus 12-month old) subjected to middle cerebral artery occlusion. In young adult groups, castrates displayed less ischemic damage as compared with intact males and castrates with testosterone replacement (Cortex: 24% in castrates versus 42% in intact versus 40% with testosterone; Striatum: 45% versus 73% versus 70%) at 22 h reperfusion. Surprisingly, supplementing testosterone in middle-aged rats to the physiologic levels ordinarily seen in young males reduced infarction (Cortex: 2% with testosterone versus 31%; Striatum: 38% with testosterone versus 68%). Testosterone effects on infarct size were blocked by the androgen receptor (AR) antagonist flutamide and further confirmed in young versus middle-aged mice. Baseline cerebral aromatase mRNA levels and activity were not different between young and middle-aged rats. Aromatase activity increased in ischemic tissue, but only in young males. Lastly, stroke damage was not different in aging aromatase knockout mice versus wild-type controls. Our findings indicate that testosterone’s effects in experimental stroke are age dependent, mediated via AR, but not cerebral aromatase.
Tags: aging, androgen receptor, aromatase, cerebral ischemia, stroke, Testosterone, Testosterone effects
20 Nov 08 Testosterone May Affect Atherosclerosis
Estrogen, Testosterone May Affect Atherosclerosis
Doctors may eventually check sex hormones to assess heart disease risk
By David March
Johns Hopkins Medicine
Naturally produced sex hormones may influence the risk and progression of atherosclerosis, or hardening of the arteries, Johns Hopkins researchers report in a recent study. The findings may help explain the increased risk men have of developing heart disease, which runs about twofold higher than women’s heart disease risk worldwide.
The study suggests that older women who produce a relatively high amount of estrogen are more likely to develop coronary artery calcium, or CAC, a component of the fatty plaque that builds up in blood vessels and hardens arteries. Older men with relatively high amounts of testosterone are also more likely to develop CAC. However, once CAC is present, higher testosterone appears to help prevent CAC from progressing too quickly in men’s arteries. These findings were presented Nov. 11 at the American Heart Association’s annual Scientific Sessions in New Orleans.
“We know many things that increase the risk of cardiovascular disease, such as high cholesterol and diabetes,” said Erin D. Michos, assistant professor of medicine at the Johns Hopkins University School of Medicine and its Heart and Vascular Institute, in an interview. “But 10 percent to 20 percent of people who get heart disease don’t have these risk factors, so we need to understand other factors that might be involved. Our results suggest that someday, in addition to testing your cholesterol and blood sugar levels to assess your heart disease risk, your doctor may want to measure your sex hormone levels.”
The study assessed whether sex hormones affect the risk of atherosclerosis using data from the Multi-Ethnic Study of Atherosclerosis, or MESA, an ongoing study that’s been tracking 6,814 patients of four different races since 2000 to determine factors that influence risk of developing cardiovascular disease. The MESA study recruited healthy people from six communities across the United States. Through a baseline assessment and regular checkups, researchers track each volunteer to learn what factors affect a person’s risk of developing cardiovascular disease or progressing once the disease develops.
For the Johns Hopkins study, researchers used data from 2,700 male and 1,646 postmenopausal female MESA participants who did not use hormone replacement therapy. At the beginning of the study, participants answered detailed questionnaires about their demographics and medical history, and they received a basic health assessment measuring their height, weight and blood pressure. Participants also received a CT scan measuring their baseline level of CAC and had their blood drawn to measure blood concentrations of various sex hormones, including estradiol, the dominant type of estrogen in women, and testosterone, the dominant sex hormone in men. About half the participants had a second CT scan 18 months later. The other half had their second scan 37 months after the initial scan.
Taking into account factors known to affect atherosclerosis risk, such as age, body mass index, blood pressure, and exercise and smoking habits, Michos and her colleagues assessed whether there was a correlation between changes in CAC between patients’ two scans and their levels of sex hormones. In women who had no baseline CAC, the researchers found that women with higher amounts of estrogen were 30 percent more likely to develop CAC by their second scan than women with lower levels of estrogen. This risk was most pronounced in women older than 65. Levels of estrogen did not seem to significantly affect whether CAC increased in women who already had CAC at baseline.
In those men who had no CAC at baseline, the researchers found that men with higher testosterone levels were 48 percent more likely to develop CAC than those with the lowest testosterone levels, with the risk greatest among men older than 55. In men who already had CAC at baseline, higher testosterone levels appeared to have a protective effect, reducing the chances that CAC measurements would increase at follow-up.
Michos added that the role that sex hormones play in cardiovascular disease is complex, with often diverse and contradictory effects. While the Johns Hopkins study looked at early atherosclerosis in the coronary arteries, sex hormones may also affect heart disease risk through other mechanisms, including influencing inflammation, blood clotting and whether blood vessels are constricted or relaxed. In the future, she and her colleagues plan to study how sex hormone levels might affect the risk of specific cardiovascular incidents, such as heart attacks and strokes, in men and women.
MESA is funded by the National Heart, Lung and Blood Institute, a member of the National Institutes of Health.
Other researchers who participated in this study are Dhananjay Vaidya, Sherita Hill Golden and Pamela Ouyang, all of Johns Hopkins; Susan R. Heckbert, of the University of Washington; and Mary Cushman, of the University of Vermont.
Tags: atherosclerosis
16 Nov 08 What is testosterone?
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Testosterone is a substance produced in the testes and in the adrenal glands that helps to build protein and is essential for normal sexual behaviour and the development of masculine characteristics such as a deep voice, broad shoulders, and hair growth.
It also affects many metabolic activities, such as production of blood cells in the bone marrow, bone formation, fat metabolism, carbohydrate metabolism, liver function and prostate gland growth. Additionally, normal testosterone levels maintain energy level, good mood, fertility and sexual desire
Low testosterone levels are typically defined as less than 300ng/dL (nanograms per decilitre) of total testosterone and less than 5ng/dL of free testosterone.
